Biomedical Sciences

Quillen College of Medicine

Jennifer Vanover Hall

Jennifer Vanover Hall

Associate Professor

 

Additional Contact Information: 

Department of Biomedical Sciences 
James H. Quillen College of Medicine 
PO Box 70577 
Office:  Bldg 1, Room 139 
Lab: Bldg 119, Room 309,310,317,318 
Phone: 423-439-6227 
Email:  halljl1@etsu.edu

EDUCATION/PROFESSIONAL EXPERIENCE

2008 - Ph.D in Biomedical Sciences/Microbiology, Quillen College of Medicine ETSU, Johnson City, TN

RESEARCH/TEACHING INTERESTS 

1.   Chlamydia trachomatis
2.   The interactions between microbial pathogens and the human host that contribute to disease pathogenesis
3.   Hormonal influences on the pathogenesis of Chlamydia trachomatis

SELECTED PUBLICATIONS

  1. Vanover Hall, J., Schell, M., Dessus-Babus, S., Moore, C.G., Whittimore, J.D., Sal, M., Dill, B.D., and Wyrick, P.B. The Multifaceted Role of Estrogen in Enhancing Chlamydia trachomatis Infection in Polarized Human Endometrial Epithelial Cells.Cellular Microbiology, 2011 13(8), 1183-1199.
     
  2. Wyrick, P.B., Schell, M., Moore, C., Whittimore, J., Vanover Hall, J., and Dessus-Babus, S. Involvement of Estrogen Receptor Beta in C. trachomatis Serovar E Infection of HEC-1B Endometrial Epithelial Cells.Proceedings of the Twelfth International Symposium on Human Chlamydial Infections, Hof bei Salzburg, Austria, 2010.

  3. Vanover, J., Kitner, J., Whittimore, J., and Schoborg, R.  Interaction of HSV-2 Glycoprotein D with the Host Cell Surface is Sufficient to Stimulate Chlamydia trachomatis Persistence.  Microbiology, 2010 156: p. 1294-1302. 
  1. Vanover, J., Sun, J., Deka, S., Kintner, J., Duffourc, M. and Schoborg, R. Herpes Simplex Virus Co-infection Induced Chlamydia trachomatis Persistence is not Mediated by any Known Persistence Inducer or Anti-chlamydial Pathway. Microbiology, 2008 154: p. 971-978.

  2. Deka, S.*, Vanover, J.*, Sun, J., Kintner, J., Whittimore, J., and Schoborg, R. An Early Event in the Herpes Simplex Virus Type-2 Replication Cycle is Sufficient to induce Chlamydia trachomatis Persistence. Cellular Microbiology, 2007 9(2): p. 725-737  *S. Deka and J. Vanover contributed equally to the work presented in this manuscript. 

  3. Deka, S., Vanover, J. Dessus-Babus, S., Whittimore, J., Howett, M., Wyrick, P., Schoborg, R. Chlamydia trachomatis enters a viable but  non-cultivable (persistent) state within herpes simplex virus type 2 (HSV-2) co-infected host cells. Cellular Microbiology, 2006 8 (1):  p. 149-62. 

  4. Schoborg, R., Vanover, J., Deka, S., Sun, J., Whittimore J., and Kintner, J. Herpes simplex virus type 2 (HSV-2) co-infection induced chlamydial persistence requires an early event in the viral replication cycle. Proceedings of the Eleventh International Symposium on Human Chlamydial Infections, Ontario, Canada, 2006.